„Bolsonaro threatens WHO exit as Brazil’s coronavirus toll soars.
47.440 Aufrufe•07.06.2020“
„Brazil’s President Bolsonaro continues to downplay coronavirus threat
102.516 Aufrufe•02.05.2020“
Kategorie: Protection
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Blood vessels, not lungs? COVID-19 might not be a respiratory disease
„Blood vessels, not lungs? COVID-19 might not be a respiratory disease“
„A group of researchers are now questioning whether COVID-19 is primarily a respiratory illness, as is widely believed.
Instead, they suggest, full-blown COVID-19 is a disease of the vascular system that preys on weakness in our blood vessels.
Does this matter in practical terms? In terms of targeting treatments in the early stage of the disease, yes it does.
But don’t we catch it from coughing and sneezing?
It’s accepted that we catch the COVID-19 via viral droplets that enter the nose, mouth or eyes, and that it presents initially as an upper respiratory tract infection.
In many to most cases, the virus appears to be defeated by the body’s immune system.
What has mystified doctors is the catastrophic second phase where patients – especially those with diabetes, cardio-vascular disease and obesity – develop a profound inflammatory response, often in the form of pneumonia.
From the start of the pandemic, so many people were dying of pneumonia, and at at an overwhelming rate, that COVID-19 was thought to be a classic respiratory disease.
Then it was found that young people were suffering blood clots and fatal strokes. Inflammation was found not only in the lungs, but in the kidneys, heart, liver, bowel and even the fingertips and toes in various patients.
Suddenly, the disease seemed deeply mysterious and even more threatening. However, when taken together, all of these symptoms pointed to an impeded blood flow.
So what is really going on?
“The concept that’s emerging is that this is not a respiratory illness alone, this is a respiratory illness to start with, but it is actually a vascular illness that kills people through its involvement of the vasculature,” said Dr Mandeep Mehra, medical director of the Brigham and Women’s Hospital Heart and Vascular Center, in a widely-reported statement.
Endothelial cells line blood vessels, like bricks in a tunnel. They form the barrier between vessels and tissues. Impaired function can lead to serious health issues throughout the body. They may be the key to COVID-19’s destructive sway. Photo: Getty
In a paper published in April, Dr Mehra and his associates found that the SARS-CoV-2 virus can directly infect and inflame the endothelial cells that line the inside of blood vessels. Damage to these cells – that play a protective role to the body – was found in all major organs.
What happens with these endothelial cells, when they become infected, appears to be the key to understanding the real nature of the disease. It is also key to that deadly second phase.
Endothelial cells form a single cell layer along the walls of all blood vessels, also lining the chambers of the heart and the lymphatic vessels, and play multiple protective roles.
The entire collection of endothelial cells – known as the endothelium – is considered to be an organ in its own right. Taken together, it weighs about a kilo, the same as the liver.
The main function of endothelial cells is to provide a barrier between the blood and the rest of the body tissues. It’s a bit like the doorman at a nightclub – in this case deciding what substances to let into the bloodstream and what to keep out.
When substances – chemicals, nutrition and white blood cells – are permitted to cross this selectively permeable layer, the endothelium then further directs them to where they are needed.
The role of the endothelium as a door-keeper is critical around the brain, restricting the passage of large molecules, toxic substances, and bacteria into the brain tissue while allowing necessary molecules like oxygen, enzymes, and hormones to go through.
But it also has a complex role in responding to pathogens – causing the sites of infection to become inflamed and hot – and to blood-clotting.
On the one hand, it makes proteins that prevent blood clotting from happening inside the blood vessel – and thereby blocking it – but also enables blood clotting at the source of bleeding.
Once infected with COVID-19, the endothelium goes awry, forfeits its ability as a guardian, and works against the body.
And this explains why the virus has so successfully invaded and damaged people from head to toe.“
Source: https://thenewdaily.com.au/life/wellbeing/2020/06/05/covid-19-respiratory-blood-vessels/ -
Rockefeller: Ziel ist jedem Menschen dieser Welt einen RFID-Chip zu verpassen. NWO Aaron Russo
„Rockefeller: Ziel ist jedem Menschen dieser Welt einen RFID-Chip zu verpassen. NWO Aaron Russo“
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Lung Doctor Analyzes George Floyd Autopsy Report (MEDICAL EXPLANATION)
„Lung Doctor Analyzes George Floyd Autopsy Report (MEDICAL EXPLANATION)“
„354.300 Aufrufe•03.06.2020
„Doctor Mike Hansen
258.000 Abonnenten
Lung Doctor Analyzes George Floyd Autopsy Report (MEDICAL EXPLANATION)
Let’s be clear..we’ve all seen the video by now. It’s obvious that these police officers killed George Floyd. The Hennepin County Medical Examiner, and the independent medical examiner hired by the family of George Floyd, Dr. Michael Baden, have concluded that his death was a homicide….but their opinion differs on the cause of death. But if both of them declared that his death was a homicide, does the cause of death really matter? (YES). I want justice for George Floyd, and that is why I’m making this video, because the medical explanation for his cause of death, is not a simple explanation. As a lung doctor, part of my job is to figure out why people can’t breathe. As an intensive care doctor, part of my job is to care for people who are on the brink of death. Like when someone can’t breathe. So when someone dies of asphyxia, as is the case of George Floyd, the determination of the cause of death is dependent on information elicited based on the investigation, which includes, the deceased personal medical history namely, autopsy, and crime scene investigation, which of course includes video evidence. Asphyxia is a Greek term that translates to “loss of pulse.”
Mechanical asphyxia involves some physical force or physical abnormality that interferes with the uptake and/or delivery of oxygen. With asphyxia, the brain doesn’t get enough oxygen, and when the pons and the medulla aren’t getting enough oxygen, they can no longer function. This means they can no longer tell the diaphragm to contract, and breathing then stops. While this happens, the heart is also not getting enough oxygen, and typically the heart pumps slower and slower until it stops. Prolonged continuous application of extreme pressure on the thorax, such as with the bodyweight of several officers, is capable of causing death. This is important, because this contributed to the death of George Floyd, in addition to the knee to the neck. The neck contains our airway, the trachea, and it also contains carotid and vertebral arteries and jugular veins. The arteries here deliver oxygenated blood to the brain, while the jugular veins allow the deoxygenated blood to flow back to the heart. So what happens when pressure is placed on the neck? Well, it depends, on a lot of different factors (amount and duration of pressure, etc). And looking at the George Floyd video, he was unconscious for more than 2 minutes with the knee still on his neck. There’s no doubt, that during this time, he took his last breath, and right around the same time, lost his pulse. By the time the EMS guy checks his pulse, I highly doubt he actually felt a pulse, because it was more than two minutes after George lost consciousness. It was obvious that when they moved George onto the stretcher, he was completely limp because he was dead. And it wasn’t until much later, did they start CPR, in the ambulance. Now let’s get to what the medical examiners had to say about this case.
Dr. Michael Baden, who did the independent autopsy says Floyd died of „asphyxiation from sustained pressure when his back and neck were compressed, with the neck pressure cutting off blood flow to his brain.” I agree with that assessment. I would also add that partial compression of the trachea, causing airway compromise, was also possible. The weight on George’s back made the work of breathing much harder for his diaphragm, and the neck pressure at the very least meant less blood (and thus oxygen) was being delivered to his brain, and less carbon dioxide could be removed from his brain. After a while, the diaphragm becomes fatigued, and no longer has the strength to contract, which means the lungs can’t get oxygen into the blood, and can’t get carbon dioxide out of the blood. And all of this caused him to lose consciousness. And probably within seconds, he lost a pulse. And despite losing consciousness, and despite losing a pulse, they continued to apply pressure on the neck, and put their weight on his back. The Hennepin County medical examiner’s office said that the cause of death is „cardiopulmonary arrest complicating law enforcement subdual, restraint, and neck compression.“ This statement doesn’t really make sense to me. But the Hennepin County release also says heart disease was an issue; the independent examiner didn’t find that. The county said that fentanyl and methamphetamine use were among „significant conditions,“ but its report didn’t say how much of either drug was in Floyd’s system or how that may have contributed. But Dr. Michael Baden got it right.
– Doctor Mike Hansen“
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Genes May Leave Some People More Vulnerable to Severe Covid-19
„Genes May Leave Some People More Vulnerable to Severe Covid-19“
Conspiracy Revelation: 6.6.2020: Most people have this blood type…
„Geneticists have turned up intriguing links between DNA and the disease. Patients with Type A blood, for example, seem to be at greater risk.“
„By Carl Zimmer
June 3, 2020
Why do some people infected with the coronavirus suffer only mild symptoms, while others become deathly ill?
Geneticists have been scouring our DNA for clues. Now, a study by European scientists is the first to document a strong statistical link between genetic variations and Covid-19, the illness caused by the coronavirus.
Variations at two spots in the human genome are associated with an increased risk of respiratory failure in patients with Covid-19, the researchers found. One of these spots includes the gene that determines blood types.
Having Type A blood was linked to a 50 percent increase in the likelihood that a patient would need to get oxygen or to go on a ventilator, according to the new study.
The study was equally striking for the genes that failed to turn up. The coronavirus attaches to a protein called ACE2 on the surface of human cells in order to enter them, for example. But genetic variants in ACE2 did not appear to make a difference in the risk of severe Covid-19.
The findings suggest that relatively unexplored factors may be playing a large role who develops life-threatening Covid-19. “There are new kids on the block now,” said Andre Franke, a molecular geneticist at the University of Kiel in Germany and a co-author of the new study, which is currently going through peer review.
Unlock more free articles.
Scientists have already determined that factors like age and underlying disease put people at extra risk of developing a severe case of Covid-19. But geneticists are hoping that a DNA test might help identify patients who will need aggressive treatment.
Figuring out the reason that certain genes may raise the odds of severe disease could also lead to new targets for drug designers.
As the pandemic gained momentum in February, Dr. Franke and his colleagues set up a collaboration with doctors in Spain and Italy who were struggling with a rising wave of Covid-19.
The doctors took blood samples from 1,610 patients who needed an oxygen supply or had to go on a ventilator. Dr. Franke and his colleagues extracted DNA from the samples and scanned it using a rapid technique called genotyping.
The researchers did not sequence all three billion genetic letters in the genome of each patient. Instead, they looked at nine million letters. Then the researchers carried out the same genetic survey on 2,205 blood donors with no evidence of Covid-19.
The scientists were looking for spots in the genome, called loci, where an unusually high number of the severely ill patients shared the same variants, compared with those who were not ill.
Two loci turned up. In one of these sites is the gene that determines our blood type. That gene directs production of a protein that places molecules on the surface of blood cells.
It’s not the first time Type A blood has turned up as a possible risk. Chinese scientists who examined patient blood types also found that those with Type A were more likely to develop a serious case of Covid-19.
No one knows why. While Dr. Franke was comforted by the support from the Chinese study, he could only speculate how blood types might affect the disease. “That is haunting me, quite honestly,” he said.
He also noted that the locus where the blood-type gene is situated also contains a stretch of DNA that acts as an on-off switch for a gene producing a protein that triggers strong immune responses.
The coronavirus triggers an overreaction of the immune system in some people, leading to massive inflammation and lung damage — the so-called cytokine storm. It is theoretically possible that genetic variations influence that response.
A second locus, on Chromosome 3, shows an even stronger link to Covid-19, Dr. Franke and his colleagues found. But that spot is home to six genes, and it is not yet possible to say which of them influences the course of Covid-19.
The Coronavirus Outbreak
Frequently Asked Questions and Advice
Updated June 5, 2020
How many people have lost their jobs due to coronavirus in the U.S.?
The unemployment rate fell to 13.3 percent in May, the Labor Department said on June 5, an unexpected improvement in the nation’s job market as hiring rebounded faster than economists expected. Economists had forecast the unemployment rate to increase to as much as 20 percent, after it hit 14.7 percent in April, which was the highest since the government began keeping official statistics after World War II. But the unemployment rate dipped instead, with employers adding 2.5 million jobs, after more than 20 million jobs were lost in April.
Will protests set off a second viral wave of coronavirus?
Mass protests against police brutality that have brought thousands of people onto the streets in cities across America are raising the specter of new coronavirus outbreaks, prompting political leaders, physicians and public health experts to warn that the crowds could cause a surge in cases. While many political leaders affirmed the right of protesters to express themselves, they urged the demonstrators to wear face masks and maintain social distancing, both to protect themselves and to prevent further community spread of the virus. Some infectious disease experts were reassured by the fact that the protests were held outdoors, saying the open air settings could mitigate the risk of transmission.
How do we start exercising again without hurting ourselves after months of lockdown?
Exercise researchers and physicians have some blunt advice for those of us aiming to return to regular exercise now: Start slowly and then rev up your workouts, also slowly. American adults tended to be about 12 percent less active after the stay-at-home mandates began in March than they were in January. But there are steps you can take to ease your way back into regular exercise safely. First, “start at no more than 50 percent of the exercise you were doing before Covid,” says Dr. Monica Rho, the chief of musculoskeletal medicine at the Shirley Ryan AbilityLab in Chicago. Thread in some preparatory squats, too, she advises. “When you haven’t been exercising, you lose muscle mass.” Expect some muscle twinges after these preliminary, post-lockdown sessions, especially a day or two later. But sudden or increasing pain during exercise is a clarion call to stop and return home.
My state is reopening. Is it safe to go out?
States are reopening bit by bit. This means that more public spaces are available for use and more and more businesses are being allowed to open again. The federal government is largely leaving the decision up to states, and some state leaders are leaving the decision up to local authorities. Even if you aren’t being told to stay at home, it’s still a good idea to limit trips outside and your interaction with other people.
What’s the risk of catching coronavirus from a surface?
Touching contaminated objects and then infecting ourselves with the germs is not typically how the virus spreads. But it can happen. A number of studies of flu, rhinovirus, coronavirus and other microbes have shown that respiratory illnesses, including the new coronavirus, can spread by touching contaminated surfaces, particularly in places like day care centers, offices and hospitals. But a long chain of events has to happen for the disease to spread that way. The best way to protect yourself from coronavirus — whether it’s surface transmission or close human contact — is still social distancing, washing your hands, not touching your face and wearing masks.
What are the symptoms of coronavirus?
Common symptoms include fever, a dry cough, fatigue and difficulty breathing or shortness of breath. Some of these symptoms overlap with those of the flu, making detection difficult, but runny noses and stuffy sinuses are less common. The C.D.C. has also added chills, muscle pain, sore throat, headache and a new loss of the sense of taste or smell as symptoms to look out for. Most people fall ill five to seven days after exposure, but symptoms may appear in as few as two days or as many as 14 days.
How can I protect myself while flying?
If air travel is unavoidable, there are some steps you can take to protect yourself. Most important: Wash your hands often, and stop touching your face. If possible, choose a window seat. A study from Emory University found that during flu season, the safest place to sit on a plane is by a window, as people sitting in window seats had less contact with potentially sick people. Disinfect hard surfaces. When you get to your seat and your hands are clean, use disinfecting wipes to clean the hard surfaces at your seat like the head and arm rest, the seatbelt buckle, the remote, screen, seat back pocket and the tray table. If the seat is hard and nonporous or leather or pleather, you can wipe that down, too. (Using wipes on upholstered seats could lead to a wet seat and spreading of germs rather than killing them.)
How do I take my temperature?
Taking one’s temperature to look for signs of fever is not as easy as it sounds, as “normal” temperature numbers can vary, but generally, keep an eye out for a temperature of 100.5 degrees Fahrenheit or higher. If you don’t have a thermometer (they can be pricey these days), there are other ways to figure out if you have a fever, or are at risk of Covid-19 complications.
Should I wear a mask?
The C.D.C. has recommended that all Americans wear cloth masks if they go out in public. This is a shift in federal guidance reflecting new concerns that the coronavirus is being spread by infected people who have no symptoms. Until now, the C.D.C., like the W.H.O., has advised that ordinary people don’t need to wear masks unless they are sick and coughing. Part of the reason was to preserve medical-grade masks for health care workers who desperately need them at a time when they are in continuously short supply. Masks don’t replace hand washing and social distancing.
What should I do if I feel sick?
If you’ve been exposed to the coronavirus or think you have, and have a fever or symptoms like a cough or difficulty breathing, call a doctor. They should give you advice on whether you should be tested, how to get tested, and how to seek medical treatment without potentially infecting or exposing others.
How do I get tested?
If you’re sick and you think you’ve been exposed to the new coronavirus, the C.D.C. recommends that you call your healthcare provider and explain your symptoms and fears. They will decide if you need to be tested. Keep in mind that there’s a chance — because of a lack of testing kits or because you’re asymptomatic, for instance — you won’t be able to get tested.
One of those gene candidates encodes a protein known to interact with ACE2, the cellular receptor needed by the coronavirus to enter host cells. But another gene nearby encodes a potent immune-signaling molecule. It is possible that this immune gene also triggers an overreaction that leads to respiratory failure.
Dr. Franke and his colleagues are part of an international effort called the Covid-19 Host Genetics Initiative.
A thousand researchers in 46 countries are collecting DNA samples from people with the disease. They are now beginning to post data on the initiative’s website.
Andrea Ganna, a genetic epidemiologist at the University of Helsinki, said that initiative’s collected data were beginning to point to a single spot on Chromosome 3 as a potentially important player.
It’s not common for genetic variants to emerge out of studies of so few people, said Jonathan Sebat, a geneticist at the University of California, San Diego, who was not involved in the new study.
“We were all hoping optimistically this was one of those situations,” Dr. Sebat said.
Previous attempts to find any genetic loci that varied significantly between sick people and healthy ones have failed. Dr. Sebat speculated that the new study succeeded because the researchers focused only on people who had respiratory failure and were clearly vulnerable to serious forms of Covid-19.
“They had the ideal cohort,” he said.
New studies, such as the one Dr. Sebat is running in California, will allow scientists to see if the two loci really do matter as much as they seem to now.
The geneticists may be able to zero in on exactly which gene in each locus affects the disease, he said. And researchers will most likely find many other genes with subtler influences on the course of Covid-19.
Genetics and the Coronavirus“
Source: https://www.nytimes.com/2020/06/03/health/coronavirus-blood-type-genetics.html
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Chemtrails – Wie eine Weltregierung unser Wetter kontrolliert
https://youtu.be/WXpeouDwp18
„Kontext: ChemtrailWikipediaVerbrecher-Lügipedia korrigiert…
Der Begriff Chemtrails, gelegentlich auch Giftwolken, und wird im Zusammenhang mit einer seit den 1990er Jahren verbreiteten und bewiesenen Verschwörungsrealität.“
„Chemtrails – Wie eine Weltregierung unser Wetter kontrolliert.“
„75.400 Aufrufe•29.04.2017“
„Zum ersten mal bemerkte ich es 2002, ein Freund machte
mich vor seinem Büro darauf aufmerksam, wie ungewöhnlich langlebig die ..Streifen sind, es ärgerte mich. Unser
einst tiefblauer Himmel war inzwischen komplett vernebelt durch künstlich geschaffene Wolken, die das Sonnenlicht
blockierten. Schließlich fand sie heraus, dass sie mit ihren Sorgen nicht allein war, auf hunderten von Internetseiten gab es Schilderungen zu den langlebigen beständigen ..Streifen.“
